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Fetal alcohol spectrum disorders (FASDs) are a group of conditions that can occur in a person who was exposed to alcohol before birth. These effects can include physical problems and problems with behavior and learning. Often, a person with an FASD has a mix of these problems. Cause and Prevention


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Fetal alcohol syndrome (FAS) is a condition that develops in a fetus (developing baby) when a pregnant person drinks alcohol during pregnancy. A syndrome is a group of symptoms that happen together as the result of a particular disease or abnormal condition.


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Fetal alcohol spectrum disorders ( FASDs) are a group of conditions that can occur in a person who is exposed to alcohol as a result of their mother drinking during pregnancy. [1]


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If your baby is exposed to alcohol during the first trimester of pregnancy it can affect how their face grows. They might have one or more of the following: small eyes. smooth philtrum (the ridges between their upper lip and nose) a thin upper lip. However, most people with FASD do not have these facial differences.


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The most severe condition in this spectrum of diseases is known as fetal alcohol syndrome (FAS). The differences in maternal and fetal enzymes, in terms of abundance and efficiency, in addition to reduced elimination, allow for alcohol to have a prolonged effect on the fetus.


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What Are Fetal Alcohol Spectrum Disorders? The term fetal alcohol spectrum disorders (FASD) refers to the wide range of physical, behavioral, and cognitive impairments that occur due to alcohol exposure before birth (also known as prenatal alcohol exposure). These impairments may appear at any time during childhood and last a lifetime. An estimated 1% to 5% of U.S. first graders have FASD.


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Introduction Prenatal alcohol exposure causes neurodevelopmental and physical alterations in the resultant offspring. The neurodevelopmental effects of in utero alcohol exposure are long-lasting affecting individuals throughout life and represent the major concerns associated with alcohol exposure during gestation (Guerri et al., 2009 ).


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While alcohol is toxic to all fetal cells and may cause defects of the kidneys, heart, lungs, eyes, ears, skin and musculoskeletal system, the developing brain is particularly sensitive. 1,2 FASD is a leading, preventable cause of intellectual disability. 3,4 Alcohol exposure at any time during pregnancy may result in damage to the developing fetal central nervous system. 5 The effects of FASD.


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Fetal Alcohol Syndrome Signs & Symptoms, In Baby, Adults & Treatment

Fetal alcohol syndrome is a condition in a child that results from alcohol exposure during the mother's pregnancy. Fetal alcohol syndrome causes brain damage and growth problems. The problems caused by fetal alcohol syndrome vary from child to child, but defects caused by fetal alcohol syndrome are not reversible.


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This is called fetal alcohol spectrum disorder (FASD). FASDs include: Fetal alcohol syndrome (FAS). This is the most severe effect of drinking during pregnancy. It may include fetal death. Infants born with FAS have abnormalities of the face. They also have growth and central nervous system (CNS) problems.


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Fetal alcohol syndrome — the severe end of the fetal alcohol spectrum disorders, which includes both neurodevelopmental disorder and birth defects caused by drinking alcohol during pregnancy


What is Fetal Alcohol Syndrome Overview, Symptoms, Causes, Diagnosis, Treatment, & Preventions

Fetal alcohol syndrome (FAS) is one of many disorders that are the direct result of a newborn whose mother consumed alcoholic beverages during the timing of her pregnancy.


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Fetal alcohol syndrome (FAS) is the severe end of a spectrum of effects that can occur when a woman drinks alcohol during pregnancy. The characteristics of FAS include growth retardation, facial abnormalities, and central nervous system dysfunction. The extreme case of alcohol-related effects is the death of the fetus and miscarriage.


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Facial and Physical Phenotype. A diagnosis of fetal alcohol syndrome (FAS) requires the presence of specific facial features, growth retardation, and evidence of central nervous system dysfunction [1-5].However, Streissguth and colleagues [] described 61 individuals with FAS spanning ages 12-40y and found that the characteristic features of FAS were less distinctive after puberty.